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肺泡上皮细胞的英文

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"肺泡上皮细胞"怎么读用"肺泡上皮细胞"造句

英文翻译手机手机版

  • alveolar epithelial cells
  • pulmonary epithelial cell

例句与用法

  • Downregulation of gap junctional intercellular communication induced by silicon dioxide in the pulmonary alveolar epithelial cen
    2刺激肺泡上皮细胞间隙连接功能下调的研究
  • Scientists at the imm used the in itro method to create lung epithelial cells known as aleolar epithelial type ii
    Imm的科学家以这种方法在体外制出了肺上皮细胞?又名ii型肺泡上皮细胞
  • It was firstly observed that cgrp plays a protective role on lung injury and attenuates glutamate - induced inhibition of pulmonary surfactant lipid synthesis . the protective mechanisms of cgrp are not mediated by blocking no production
    并进一步观察到rp具有抗iu所致肺泡上皮细胞损伤的保护作用,可减轻o对ps脂质合成的抑制,但cgrp的作用并不是通过阻断o激活nos ,使no生成减少而实现的。
  • Conclusions electron microscopic obser vations show that the alveolar epithelial cells , endothelial cells and basement membrane are all injured , interstitial macrophages hyperpplasia , mast cells infiltration and degranulation , electron - dense deposits at alveolar wall are present
    结论电镜观察可见特发性肺纤维化时肺泡上皮细胞、血管内皮细胞及基底膜均有损伤,并可见间质巨噬细胞增生,肥大细胞浸润及脱颗粒,肺泡壁电子致密物沉积。
  • Mechanism of glutamate - induced inhibition of pulmonary surfactant lipid synthesis ? mda receptor ( nmdar ) antagonist mk - 801 could reverse glutamate - induced inhibition of [ 3h ] - choline incorporation in lung explants ; ? he expression of nmdar1 subtype existed in alveolar type ii epithelial cells ; ? he effect of glu on pc synthesis required l - arginine in cultured lung explants , nos activity and no production were increased significantly in lung explants with glu treatment ; and mk - 801 and nos inhibitor l - nna could block the increase of nos activity and no production induced by glu ; l - nna also could reverse the inhibition of [ 3h ] - choline incorporation induced by glu ; ( 4 ) mda content increased and sod level decreased in cultured lung explants of glu treament groups , mk - 801 and l - nna could reverse these effects ; ( 3 ) mk - 801 , l - nna could block the release of ldh induced by glu
    谷氨酸对肺表面活性物质脂质合成抑制的机制研究nmda受体拮抗剂mk - 801能逆转glu所致肺组织[ ~ 3h ] -胆碱掺入减少的效应;肺泡上皮细胞有nmda受体nmdar1亚单位的表达; glu对ps脂质合成的影响依赖l -精氨酸的存在, glu可使肺组织匀浆中nos活性、 no合成量升高; mk - 801 、 nos的抑制剂l - nna均可阻断glu所致的这些效应; l - nna可逆转glu所致肺组织[ ~ 3h ] -胆碱掺入减少的效应; glu可使肺组织匀浆中mda硕士学位论文中文摘要含量增高、 sod水平下降。 mk七0 、 l nna能阻断q所致的这些效应; mk80 、 l nna还能阻断gill诱导的ldh释放增多的效应。
  • Results the major morphologic changes were as follows : histologically , alveolar inflammation and interstitial fibrosis were observed . electron microscopic findings were : 1 . alveolar type i cells were degenerated 、 broken - down and desquamated , endothelial cells were swelled , with inter cellular tight junction shortened , alveolar type ii cells hyperplastic , basement membrane thinned and deformed ; 2 . alveolar macrophages and interstitial macrophages were hyperplastic ; 3 . mast cells were infiltrated and degranulated ; 4 . electron - dense deposits were present at alveolar wall ; 5 . myofibroblasts 、 fibroblasts 、 collagen and basement membrane like material were hyperplastic
    电镜观察可见: ( 1 ) i型肺泡上皮细胞变性、崩解和脱落,内皮细胞肿胀,细胞间紧密连接短小, ii型肺泡上皮细胞增生,基底膜变薄和破坏; ( 2 )肺泡巨噬细胞、间质巨噬细胞增多; ( 3 )肥大细胞浸润并见脱颗粒现象; ( 4 )肺泡壁电子致密物沉积; ( 5 )肌纤维母细胞、纤维母细胞、胶原原纤维及基底膜样物质增生。
  • We also investigated the pathological changes of mouse liver , thymus and cerebrum cortex challenged by so2 inhalation by in vivo tests . we studied the apoptotic induction on mouse spleen cells and cytotoxicity of human embryo lung fibroblasts of so2 derivatives by in vitro tests . in vivo tests of sulfur dioxide inhalation showed : ( 1 ) effects on mouse lung of so2 challenge : we found no significant apoptotic changes induced by so2 inhalation but obvious pathological changes of lung with vacuolating of osmiophilic multilamellar bodies which maybe related with the decrease of surfacant and decrease of microvillus of type ii alveolar cells ; we also found thickening of part of basement lamina between type i alveolar cells and capillary endothelium cells which may inhibit the dispersion of oxygen and contribute to lung dysfunction
    二氧化硫熏气染毒的体内实验结果表明,在本次实验的浓度范围内( 56mg m ~ 3 、 112mg m ~ 3 、 168mg m ~ 3低、中、高三个浓度) : ( 1 )通过透射电镜、 dna凝胶电泳分析和流式细胞分析发现二氧化硫吸入染毒一周对小鼠肺脏没有明显的凋亡诱导作用,但通过透射电镜观察发现二氧化硫可引起肺脏明显的超微结构改变,引起型肺泡上皮细胞板层体空泡化,微绒毛减少,线粒体致密化或肿胀变性;肺泡血管内皮细胞和型肺泡上皮细胞之间基膜增厚,使氧气弥散功能出现障碍,从而降低肺功能。
  • Transplantable aleolar epithelial type ii cells may also one day be helpful in the treatment of other lung diseases including chronic obstructie pulmonary disease ( copd ) , the fourth leading cause of death in the united states , claiming the lies of 122 , 283 americans in 2003 , and asthma , wetsel said
    可移植ii型肺泡上皮细胞有一天可能会有助于治疗其它肺部疾病包括慢性阻塞性肺病( copd )及哮喘,慢性阻塞性肺病是美国第四大致死疾病,仅在2003年就夺去了122 , 283人的生命。
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