rolandic造句

• Rolandic seizures are usually brief, lasting for 1 3 min.
• This is due to the varying locations of the brain the Rolandic.
• Learning can remain unimpaired while a child is afflicted with Rolandic epilepsy.
• It is classified amongst benign idiopathic childhood focal epilepsies such as rolandic epilepsy and Panayiotopoulos syndrome.
• These studies have also found that there are anatomical differences in the Rolandic operculum and arcuate fasciculus.
• Speech production activations begin prior to verbal response in the peri-Rolandic cortices ( postcentral gyri ).
• Patients with frontal spikes were significantly older than patients with spikes on rolandic, parieto-occipital, or calcarine sulci.
• In Italy, Luigi Rolando carried out lesion experiments and performed electrical stimulation of the brain, including the Rolandic area.
• They show underactivity in the ventral premotor, Rolandic opercular and sensorimotor cortex bilaterally and Heschl s gyrus in the left hemisphere.
• Another comparison of scans anterior forebrain regions are disproportionately active in stuttering subjects, while post-rolandic regions are relatively inactive.
• The role of ventral peri-Rolandic cortices in speech motor functions has long been appreciated ( Broca's area ).
• Some childhood epilepsy syndromes are included in the unknown cause category in which the cause is presumed genetic, for instance benign rolandic epilepsy.
• However, some children may present with independent autonomic seizures or seizures with mixed Rolandic-autonomic manifestations including emesis as in Panayiotopoulos syndrome.
• Despite prominent hypersalivation, focal seizures with primarily autonomic manifestations ( autonomic seizures ) are not considered part of the core clinical syndrome of Rolandic epilepsy.
• Rolandic epilepsy may present with atypical manifestations such early age at onset, developmental delay or learning difficulties at inclusion, other seizure types, atypical EEG abnormalities.
• The multifocal nature of epileptogenicity in Panayiotopoulos syndrome has been also documented with MEG, which revealed that the main epileptogenic areas are along the parietal-occipital, the calcarine, or the central ( rolandic ) sulci.
• Patients whose surgeon uses cortical stimulation mapping to assess the anatomy and function of rolandic areas have a greater chance and faster rate of regaining baseline function post-operatively than those who undergo surgeries that avoid this technique.
• The expression of ELP and the fourth sub-unit ( ELP4 ) in particular is the cause of Rolandic epilepsy and possibly other cognitive impairment later in life if the condition is severe enough or if it is not treated effectively.
• Less serious examples are benign rolandic epilepsy ( 2.8 per 100, 000 ), childhood absence epilepsy ( 0.8 per 100, 000 ) and juvenile myoclonic epilepsy ( 0.7 per 100, 000 ).