The study revealed novel characteristics of a protein , called ns1 , that activates a key pathway in the virus ' s reproduction 该研究显示出了ns1蛋白质的新特性,它可以激活病毒复制的关键步骤。
Still lacking has been a system to sustain replication of noel irus isolates from patients using nontransformed hepatocytes 目前依然没有利用患者非突变肝细并能维持足够的病毒复制的培养体系。
For instance , they might construct a compound that inhibits the active site of an enzyme crucial to viral reproduction 举例来说,他们可以设计某个化合物,作用在病毒复制时不可或缺的酵素活性区,而加以抑制。
When sera from patients infected with different hc strains were added to the hepatocyte culture system , iral replication occurred and new irus particles were produced 发现当hc携带者的血清加入该培养系统后,仍有大量病毒复制繁殖。
If you see reactivation , chb in patients that might cause liver failure that early suppression of virus and progression of liver failure 但一旦激活了病毒复制,早期病毒受抑制的慢乙肝患者后期慢乙肝患者有可能逐步发展为肝功能衰竭。
All facts suggest that the ns5 protein plays an important role in viral rna replication , so it maybe a target to prohibit the viral replicating 鉴此, ns5可以成为阻断或抑制病毒复制的靶标,而目前国内外尚无以denns5作为抗病毒靶标的研究报告。
First is the establishment gram through with virus s competition , consumes polymerase which the viral duplication needs , thus lastingly suppresses the viral duplication , prevents 首先是安立克通过和病毒的竞争,消耗掉病毒复制所需要的聚合酶,从而持久地抑制病毒复制,阻止
Once the membranes are fused , the virus dumps its genetic content into the healthy human cell ' s interior , hijacking the cell ' s replication machinery to clone itself 一旦细胞膜被融解,病毒就会将自身的基因物质倾注到健康的人体细胞中,并夺取人体细胞能量来进行病毒复制。
Interestingly , cultured hepatocytes responded to iral replication by displaying signs of distress and cell death and by expressing interferon - beta , a cellular antiiral , in an effort to control the infection 有趣的是,培养系统同时也通过应激或细胞凋亡来对抗病毒复制,并且表达干扰素,产生细胞毒性,控制炎症进展。
First is the annlic establishment gram through with virus s competition , consumes polymerase which the viral duplication needs , thus lastingly suppresses the viral duplication , prevents the disease the progress 通过和疱疹病毒的竞争,消耗掉疱疹病毒复制所需要的聚合酶,从而使疱疹病毒失去繁衍所需要的基础物质,让疱疹病毒在身体里无法复制最终彻底死亡。