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d-galactose

"d-galactose"的翻译和解释

例句与用法

  • Effects ofpolygala tenuifolia willd on sod activity of rbc and gsh - px activity of liver tissue inaging mouse caused by d - galactose
    肝组织谷胱甘肽过氧化物酶活性影响的实验研究
  • Effect of active component of four types of chinese herbs on aldose reductase activity and non - enzymatic protein glycation in d - galactose induced rats
    半乳糖诱导大鼠醛糖还原酶活性和蛋白非酶糖化的作用
  • Influence of d - galactose on rat spatial learning memory behavior as well as cerebral hippocampal electrophysiology and synaptic morphology
    半乳糖对大鼠空间学习记忆行为与脑海马结构电生理以及突触形态学的影响
  • Effect of chinese drug lishi no . 5 on the ultrastructure of hippocampal neurons and expression of insulin receptor substrate in d - galactose induced aging mice
    半乳糖老化小鼠海马超微结构及胰岛素受体底物表达的影响
  • Effect of electroacupuncture on changes of amino acid neurotransmitters in the hippocampus of rats with spatial learning and memory impairment induced by d - galactose
    半乳糖所致空间记忆障碍大鼠海马氨基酸递质改变的影响
  • The hemagglutinating activity of amphioxus skin mucus was decreased after heating at 60 for 30 min , and was almost lost upon heating at 100 for 30 min . the hemagglutinating activity of the skin mucus remained stable in the ph range from 6 to 10 , but it was inactivated at ph above 10 or below 6 . the hemagglutinating activity in the skin mucus is inhibited by d - galactose , which is same with the hemagglut inating activity of the humoral fluids of amphioxus
    在60保温30分钟后,凝集素的凝集活性下降,在100保温30分钟后,凝集素凝集活性几乎完全消失;在ph6 - 10的范围内,凝集素的凝集活性稳定, ph大于10小于6的条件下,凝集素的凝集活性消失;文昌鱼体表粘液中的凝集素受d -半乳糖苷的抑制。
  • Reviewing the application and development of d - galactose induced aging model , and different measurements in different records , we summarized the establishment and theory of d - galactose induced aging model , furthermore , we described the measurements from ethology , biochemstry . morphology and molecular biology in this review
    鉴于该模型近年来的运用和发展,且众文献中的检测指标各有侧重,本文拟对该模型构建方法和原理以及检测指标从行为学水平,生化水平,形态学水平,分子生物学水平等方面由浅入深进行综述。
  • Methods subacute senile mouse model was established by injection of d - galactose . the splenic cells were isolated from mice and cultured in vitro following cona stimulation . expression of cd 137 on splenic t cells in the model and normal control groups were compared with each other by flow cytometry method at various cultural times after cona stimulation
    方法取5周龄balb c雄性小鼠,颈背部皮下注射d -半乳糖,分别于造模型后一到五个月取脾脏淋巴细胞,同时取自然衰老小鼠脾脏淋巴细胞,在培养的第0 、 24 、 48 、 72 、 96 、 120 、 144h收集细胞,用流式细胞仪分析脾脏t细胞cd137的表达阳性率。
  • Conclusions ( 1 ) subacute senile mouse model can be used in immunosenesence reseach . ( 2 ) cd 137 may be an marker of aging . ( 3 ) the protection to aicd of cd137 decreased , indicate the function of cd137 was unusual . ( 4 ) the effect of d - galactose can modelling the different stage of aging . ( 5 ) the decrease of cd 137 expression on t cell from subacute senile mouse model and the aging mouse were due to the declining of their mrna . ( 6 ) the expression of cd 137 on t cells from from subacute senile mouse model and the aging mouse have a time dependently derease and the peak of cd 137 expression appeared earlier while the aging keep going on
    结论( 1 ) d -半乳糖致亚急性衰老小鼠模型t细胞cd137分子表达变化规律与自然衰老小鼠相似,造模两个月后的大剂量组小鼠即可用于衰老个体cd137分子的相关研究。 ( 2 )模型小鼠cd137分子表达随衰老的发生发展呈规律性变化,提示该分子可能为t细胞衰老的分子标志。 ( 3 )即使在衰老状态下,机体对cd137分于的表达依然具有调控作用,提示兔疫系统的代偿能力仍然存在。
  • Results : it was about genomic polymorphism analyses of 5 parts of brain ( brain regions ) , composed of cortex , hyppocampus , sriatum , hypothalamus and cerebellum , in variety of the sd - rats . 1 , it was showed that special genomic rapds were different between the sd - rats at ages of 3 months old , 6 - 8 months old , 18months old and 24 months old , and there was some correlation between sd - rat " ages of months old adding up and increase of brain regions with special rapds . 2 , special genomic rapds of the brain aging sd - rat at the age of 24 months old was the same as that of the brain aging sd - rat induced by - d - galactose from the age of 3months old
    结果对不同生理状态( 3月龄, 6 - 8月龄, 18月龄, 24月龄)下的脑皮层、海马、纹状体、下丘脑、小脑等五脑区进行基因组dna多态性分析,发现:不同月龄大鼠脑区的基因组特异性rapd分子标记不同,月龄的增长和脑区基因组特异性rapd分子标记的增多呈一定的相关性;对不同实验处理( - d -半乳糖致脑衰老,花粉、复方氨基酸、吡拉西坦抗脑衰老)后的五脑区进行基因组dna多态性分析,发现: - d -半乳糖致脑衰老与24月龄自然脑衰老脑区的基因组特异性rapd分子标记相似,花粉、复方氨基酸、吡拉西坦抗均不同程地改变了- d -半乳糖致脑衰老脑区基因组特异性rapd分子标记。
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