The actin cytoskeleton , a dynamic network of intracellular proteinaceous structural elements participates in many physiological activities in plant cell . the actin cytoskeleton polymerization and depolymerization is regulated by a number of actin - binnding protein rapidly and spatio - temporally 植物细胞中的微丝骨架，是细胞内的一个由蛋白质构成的动态网络结构，参与胞内诸多的重要的生理活动。
. . . nearly all aspects of cell life are regulated by reversible protein phosphorylation . some examples include metabolic processes , gene regulation , cell cycle control , transport and secretory processes , the organization of the cytoskeleton , and cell adhesion 蛋白可逆磷酸化几乎可以调节所有的细胞活动，包括代谢过程、基因表达调控、细胞周期调控、运输和分泌过程、细胞骨架的形成和细胞粘连。
Experiments showed that when a cell ' s cytoskeleton was incapacitated by toxins , some proteins were no longer able to move toward the immune synapse , suggesting that movements of cytoskeletal filaments allow cells to control when and where the proteins accumulate at the synapse 实验显示，当细胞骨架受到毒素破坏而无法运动时，有些蛋白质就再也无法朝免疫突触移动，这意谓著，细胞藉由细胞骨架纤维的运动，来控制蛋白质在突触聚集的时机与位置。
The results suggested : the differential gene expression in the adrenal gland under acute hyperglycemia / hypoglycemia was related to blood glucose ; these genes might regulate blood glucose by influencing the metabolism and secretion of hormones , cytoskeleton constitution and signal transduction in the adrenal gland ; bbs4 gene might participate in glucoregulation via the activities of adrenal medulla ; the differential genes expressed under acute vs chronic hyperglycemia might play a basic role of the structural and functional changes in the adrenal gland , lead to the process of chronic complications of diabetes or accommodate to the status of hyperglycemia 结果提示：肾上腺基因差异性表达和血糖变化有关；在急性高低血糖情况下，这些基因可能通过影响肾上腺内激素的分泌和代谢、细胞骨架构成和信号转导来调节血糖； bbs4可能参与血糖调节，其作用可能和肾上腺髓质的活动有关；慢性高血糖情况下的肾上腺差异表达不同于急性高血糖，这种表达差异可能与肾上腺慢性的结构和机能改变相关，或参与了糖尿病慢性并发症的发生发展、高血糖状态的调节。
In this review , the present status of the molecular and physiological mechanism of the differentiation and development of cotton fiber is discussed , in which the molecular and physiological mechanism of the producing of the cell turgor , loosening of the cell wall , synthesis of the composition molecules and their incorporation into cell wall , signal of the starting of secondary wall thickening , cellulose biosynthesis , and layout of cellulose microfibril regulated by cytoskeleton are introduced in detail 摘要本文综述了棉纤维分化、发育的分子生理机制的研究现状，著重讨论了棉纤维细胞膨压的产生、细胞壁的松弛、结构分子的合成和加入、次生壁增厚的启始信号、纤维素的生物合成和细胞骨架系统控制纤维素沉积等机制。
It may have a preventional function from alzheimer ' s disease . molecular biological studies are shown that apoe isoform has a isoform - specific role to neurodegeneration . apoe3 and apoei aid to sustain the stabilization of cytoskeleton protein tau , while apoe4hyperphosphorylates tau to destabilize the microtubule protein , which will make neurocyte easy to form neurofibrillary tangles in ad 分子生物学研究发现， apoe异构体对神经细胞退化表现出异构体特异作用， apoe _ 3和apoe _ 2有助于维持微管的稳定性，而apoe _ 4则使tau蛋白超磷酸化，破坏微管的稳定性，使神经细胞易于形成神经纤维缠结。
The fusion gene ofgfp : mtn ( mtn is binding domain of microfilament binding protein talin , which can show the microfilament in living cell ) was transferred to arabidopsis thaliana . stable expression of gfp - mtn can visualize the actin cytoskeleton in different types in living cells without affecting cell morphology and function 将gfp : mtn ( mtn是微丝结合蛋白talin的微丝结合域，可以显示活体细胞中微丝的结构)导入拟南芥后，发现表达的融合蛋白能够标记微丝骨架，同时又不影响植物细胞的正常形态和功能。
